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During thistransition phase FOXP3 expression is reduced, Treg inhibitory function ceases, and theRORgammat gene expression begins to take over. This may lead to posttraumatic stressdisorder and certainly causes anxiety and an altered mental state.

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A variety of evidence supports the conceptthat central auditory pathways participate maladaptivelyin the pathophysiology of tinnitus: (1) tinnitus or periph-eral origin can persist after recovery of cochlear func-tion, labyrinthectomy, or eighth-nerve neurectomy; (2)unilateral tinnitus can be suppressed by either homo- orcontralateral noise; (3) damage to the inner ear producessubstantial structural, neurochemical, and physiologicneural changes in the auditory pathways of the brainstemand cerebrum, including enhanced sound-driven activity,increased spontaneous neural activity, altered neural fi r-ing (such as bursting discharges and neural synchrony),reorganization of the tonotopic representation of fre-quency in the brainstem and cerebral cortex, and exten-sion of spontaneous cortical activity associated with tinni-tus into nonsensory areas (Saunders, 2007). They actin a manner similar to etoposide buy modafinil in us but interact witha different enzyme (DNA topoisomerase-1).